The accepted knowledge is that Diabetes destroys gradually over years. Ketosis Prone Type 2 diabetes is an acute form of type 2. This type 2 can reach fasting blood sugars of 300 or higher in months. This blog brings together all the documentation that I could find in the world and my speculation of what it means for KPD’s in specific and diabetics in general. I ask you to leave your stories about what happened to you so that we can all gain a better understanding of what we are dealing with.

Wednesday, November 28, 2012

The KPT2 point of view of Diabesity Pt 1

Recently, I was at a diabetes conference representing the Michigan Minority Health Coalition. There were many presentations that dealt with ways to slow or reverse the diabetes epidemic. Typically, they involved ways to get people to control what they ate and getting exercise. I was sitting with a group of researchers and they, unfortunately, asked me what I thought. I went on my usual depressing rant.

I’m not seeing any of this as being useful and the data seems to bear me out. Diabetes continues to increase across the US. We are pouring more time and resources into it with little or no effect.

One of the reasons for my negativity is that Ketosis Prone Type 2 diabetes seems to contradict most of the standard wisdom we know about diabetes.

First of we are the Type 2’s that can and do go DKA. Though people think of us as relatively rare, we are increasingly being recognized as a problem in emergency medicine as we force the costs of treatment up worldwide..

Not only do we go DKA, we also can go into remission. Taking no medications just simply using diet, some exercise and no weight loss, we can return to near normal blood sugars for years.

What is even odder about this DKA and remission is how close they occur together. Two weeks after a DKA episode, where blood sugar readings were nearly off the charts, the person can start experiencing hypos. Remember, KPT2 is classified by the ADA as a Type 1, specifically Type 1b. Yet, typically after six months, the person doesn’t even need insulin or much of anything else. As I’ve noted in previous posts, this recovery is much too fast to be attributed to beta cell regeneration. Even if you are inclined to believe that there might be super fast regeneration, I detail in past posts, how I was able to manipulate my pancreatic output in days by taking an anti-inflammatory.

It is well documented that weight is not a major factor. In fact, KPT2s who are overweight do better than those who have, so called “healthy weights”. It is obvious that the last recommendation that a doctor would make is for that person to lose weight.

What if we were the prevalent diabetes of the world. How would the rules be different? 

Stay tuned for part 2.