The accepted knowledge is that Diabetes destroys gradually over years. Ketosis Prone Type 2 diabetes is an acute form of type 2. This type 2 can reach fasting blood sugars of 300 or higher in months. This blog brings together all the documentation that I could find in the world and my speculation of what it means for KPD’s in specific and diabetics in general. I ask you to leave your stories about what happened to you so that we can all gain a better understanding of what we are dealing with.

Monday, December 31, 2012

Traditional foods ... again

This popped up a few days ago and is very much in line with what I've been saying about diet and food.  Here.
This is a study done in Lebanon (KPD is well-documented in the Middle East). It should be noted that there was one other diet that was examined but it showed no association with diabetes. This diet would be close to a low carb diet.

The findings of this study demonstrate direct associations of the Refined Grains & Desserts and Fast Food patterns with T2D and an inverse association between the Traditional Lebanese pattern and the disease among Lebanese adults. 
 Dietary patterns and odds of Type 2 diabetes in Beirut, Lebanon: a case - a case study

What should be noted is that this diet maybe safe now but it too is subject to the substitution of ingredients as well so complacency, as regards ones diet, should be guarded against.

Diabesity 2

This is my rant taking my type of diabetes as the major form in the world. I make no apologies to anyone. We are forgotten but we are important because it gives a view of diabetes that is different and necessary.

 Diabetes is ultimately caused by the failure of the immune system due to inflammations introduced by environmental toxicity. An environment is toxic if it does not support the health of an organism. This can be toxins that are viral, bacterial, physical or chemical and it may also be the case that the environment is toxic if it doesn’t supply the necessary components for health. It is also true that almost anything can be toxic above a certain threshold. This is where long term genetic adaptation comes in.

Over time a successful organism will change in ways to suit that environment. It will develop the mechanisms to recognize then offset or neutralize toxins. What is important is the very systems that must cooperate are in some ways disrupted and can not complete their functions leading to more inflammation and less cooperation. We see autoimmune diseases, sensitivities and deficiencies as part of this.

The net effect of this is a some what continuous suboptimal functioning which forces the body to compensate. The outcome of all this is what we call “diabetes”. There are all kinds of toxins out there but the one I’m going to concentrate on has to do with diet.

Why diet, two things: diseases of civilization and obesity. “Diseases of civilization” have been noted for centuries and is caused when some previously unexposed culture adopts our diet. This occurred largely before we had the scientific sophistication to develop most of the chemicals that people say plague our environment today. It also precedes the wide spread manipulation of foods, both plants and animals, by industrial systems to increase yields and profits. The rise of diabetes and obesity seem to be moving hand in hand. Weight gain, no matter whatever else it is about, is about eating and eating is the port of our major exposure to the environment.

 I was an athlete and many of the people I’ve corresponded with were as well. A change of less workouts, for whatever reasons, but still the same eating pattern turned quickly into the abrupt onset of diabetes. How can you go quickly from athleticism to diabetes? On the face of it, this would seem impossible but it isn’t. It very simply takes 6 months and a person can be DKA, if they continue on their same diet, which has been the accepted healthy diet.

One of the reasons for this is that muscle does not protect against high blood sugars as well as fat. The continual insult to endocrine tissues does not abate. When a person has finished a work out and carbohydrates loads, once the muscles have taken up all they can, the body will evince higher blood sugars that will continue until the next round of exercise. This is a creeping effect, continual insult but with most of the blood sugar problem generally being contained by the consistent exercise of muscle. This will keep the blood sugars down most of the time but will not ameliorate the effects of the continual inflammation of endocrine and other tissues.

Maybe, at one time this would occur but this is inflammation. Continual exercise keeps the dog from the door but never reduces the effects of inflammation that has already been introduced. It is still there and all the disruptions in signals between the organs is still present due to the continual post prandial challenges and, probably, other continual environmental challenges

Wednesday, November 28, 2012

The KPT2 point of view of Diabesity Pt 1

Recently, I was at a diabetes conference representing the Michigan Minority Health Coalition. There were many presentations that dealt with ways to slow or reverse the diabetes epidemic. Typically, they involved ways to get people to control what they ate and getting exercise. I was sitting with a group of researchers and they, unfortunately, asked me what I thought. I went on my usual depressing rant.

I’m not seeing any of this as being useful and the data seems to bear me out. Diabetes continues to increase across the US. We are pouring more time and resources into it with little or no effect.

One of the reasons for my negativity is that Ketosis Prone Type 2 diabetes seems to contradict most of the standard wisdom we know about diabetes.

First of we are the Type 2’s that can and do go DKA. Though people think of us as relatively rare, we are increasingly being recognized as a problem in emergency medicine as we force the costs of treatment up worldwide..

Not only do we go DKA, we also can go into remission. Taking no medications just simply using diet, some exercise and no weight loss, we can return to near normal blood sugars for years.

What is even odder about this DKA and remission is how close they occur together. Two weeks after a DKA episode, where blood sugar readings were nearly off the charts, the person can start experiencing hypos. Remember, KPT2 is classified by the ADA as a Type 1, specifically Type 1b. Yet, typically after six months, the person doesn’t even need insulin or much of anything else. As I’ve noted in previous posts, this recovery is much too fast to be attributed to beta cell regeneration. Even if you are inclined to believe that there might be super fast regeneration, I detail in past posts, how I was able to manipulate my pancreatic output in days by taking an anti-inflammatory.

It is well documented that weight is not a major factor. In fact, KPT2s who are overweight do better than those who have, so called “healthy weights”. It is obvious that the last recommendation that a doctor would make is for that person to lose weight.

What if we were the prevalent diabetes of the world. How would the rules be different? 

Stay tuned for part 2.

Wednesday, October 3, 2012

Recognition for KPT2D

It’s been a bit of a while since I wrote on this blog. It isn’t that I’ve lost interest but it seems as if the world has. I continually monitor for papers on it but there really isn’t much.

I have taken the view that this blog is not personal but is a way of gathering as much scientific and medical information as possible on KPD so that sufferers and their friends and family will have a source to explore and maybe gain hope or understanding.

I present this information in this light. This is from the Diabetes in Control Mastery Series. It is based on the work of researchers out of Athens, Greece. The book is about diabetic emergencies but it takes the time to illuminate KPD because of its recognition as a large component in the rising rate of DKA worldwide.

There isn’t one word here that you won’t find in this blog. It is, however, important, that we get recognition anyway we can. This sort of thing, hopefully, will bring more money and more research.